Newsletter # 124

In vivo studies
Consistently induces significant cellular infiltration, including granulocytes (GRAN also known as Polymorphonuclear), lymphocytes (LYMP) and monocytes (MON)—mimicking complex tissue-level inflammation.
Elevated pro-inflammatory cytokines offer precise molecular readouts to monitor therapeutic modulation.
The model responds robustly to corticosteroid treatment. Dexamethasone significantly reduces inflammation, normalizing immune cell counts and cytokine levels. This effect is often visualized as a clear drop in the "Dexamethasone-treated" group (grey column) compared to the placebo-treated group (blue column), with the inflammation-free control group (orange column) providing a baseline reference. With its well-defined cellular and molecular parameters, and strong alignment with clinical inflammation profiles, the carrageenan air pouch model is an ideal system for screening and validating novel anti-inflammatory agents.
With its well-defined cellular and molecular parameters, and strong alignment with clinical inflammation profiles, the carrageenan air pouch model is an ideal system for screening and validating novel anti-inflammatory agents.
-
Values are expressed as mean ± standard error of the mean (SEM). *, *** denote statistically significant differences between treatment groups, as compared to Carrageenan + placebo-treated group.
Orange column: Non-inflamed control group,
Blue column: Carrageenan + placebo-treated group,
Grey column: Carrageenan + dexamethasone-treated group
Leukocyte Subpopulations: GRAN: Granulocytes (Polymorphonuclear cells), LYMP: Lymphocytes, MON: Monocytes
-
We look forward to hearing from you.
Get in touch
